rabbit polyclonal anti phospho ampk t172 (Cell Signaling Technology Inc)
Cell Signaling Technology Inc is a verified supplier 
Cell Signaling Technology Inc manufactures this product 
86
Structured Review
Cell Signaling Technology Inc
rabbit polyclonal anti phospho ampk t172
Rabbit Polyclonal Anti Phospho Ampk T172, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/phospho+ampk+t172/pmc13052180-62-65-73
Average 86 stars, based on 1 article reviews
Rabbit Polyclonal Anti Phospho Ampk T172, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/phospho+ampk+t172/pmc13052180-62-65-73
Average 86 stars, based on 1 article reviews
rabbit polyclonal anti phospho ampk t172 - by Bioz Stars,
2026-10
86/100 stars
Images
1) Product Images from "CpG oligodeoxynucleotide reduces PrP Sc accumulation and prolongs survival in prion-infected mice"
Article Title: CpG oligodeoxynucleotide reduces PrP Sc accumulation and prolongs survival in prion-infected mice
Journal: Molecules and Cells
doi: 10.1016/j.mocell.2026.100335
Figure Legend Snippet: CpG ODN induces AMPK signaling and attenuates phosphorylated p62 accumulation in 22L scrapie-infected mice. (a) Western blot analysis of brain lysates at 170 dpi showing expression levels of p-AMPK T172, AMPK, p-ULK1 S555, ULK1, p-p62 S403, p62, ATG12–5, and LC3 I/II in brains of 22L scrapie-infected mice with or without CpG ODN at 170 dpi. (b) Relative intensity of p-AMPK, p-ULK, p62, p-p62, ATG12–5, and LC3-II represented as bar graphs (mean ± S.E.M, n = 6). Statistical significance was determined by 1-way ANOVA with Tukey’s post hoc test. * P < .05, ** P < .01, *** P < .001. NS: not significant.
Techniques Used: Infection, Western Blot, Expressing
Figure Legend Snippet: CpG ODN reduces PrP Sc accumulation and activates AMPK-associated signaling in 22L scrapie-infected neuronal cells. (a and b) Immunoblot analysis of (a) PrP Sc and total PrP, and (b) TLR9, p-AMPK T172, total AMPK, p-ULK1 S555, total ULK1, ATG12–5, total p62, and LC3 I/II in 22L scrapie-infected neuronal cells (ZW-22L) treated with CpG ODN (0, 1, or 3 µM) for 6 hours. For PrP Sc detection, equal amounts of protein were incubated with proteinase K (2 µg/ml) for 1 hour. (c) Densitometric quantification of p-AMPK, p-ULK1, ATG12–5, total p62, and LC3-II levels. Data are presented as mean ± S.E.M ( n = 3). (d) Immunoblot analysis of PrP Sc , p-AMPK T172, and total AMPK in ZW-22L cells treated with CpG ODN (3 µM, 6 hours) in presence or absence of the TLR9 antagonist ODN 2088 (5 µM, 7 hours). Data represents 3 independent experiments ( n = 3). Statistical significance was determined by 1-way ANOVA with Tukey’s post hoc test (* P < .05, *** P < .001).
Techniques Used: Infection, Western Blot, Incubation
Related Articles
Control:Article Title: Cyanine dye conjugates of a 2’-deoxycytidine-based auto- and mitophagy activator extend Caenorhabditis elegan s lifespan Article Snippet: .. The following antibodies were used: Tuberin/TSC2 (3612, Cell Signaling Tech., USA), phospho-Tuberin/TSC2 (Ser1387) (5584, Cell Signaling Tech., USA), ULK1 (8054, Cell Signaling Tech., USA), phospho-ULK1 (Ser555) (5869, Cell Signaling Tech., USA), phospho-ULK1 (Ser317) (89267, Cell Signaling Tech., USA), phospho-ULK1 (Ser757) (6888, Cell Signaling Tech., USA), AKT (9272, Cell Signaling Tech., USA), phospho-Akt (Thr308) (9275, Cell Signaling Tech., USA), AMPK (2532, Cell Signaling Tech., USA), other:Article Title: Increased regional activity of a pro-autophagy pathway in schizophrenia as a contributor to sex differences in the disease pathology Article Snippet: Phospho-AMPK T172 , Article Title: Design, synthesis and biological evaluation of benzimidazole carbamide derivatives - potential autophagy inducers. Article Snippet: Herein, we report a convenient synthetic approach and an in vitro evaluation of benzimidazole carbamide derivatives as potential autophagy activators.. Two compounds exhibited autophagy-inducing activity at concentrations ≥100 μM.. The most favorable activity–toxicity profile was observed for a benzimidazole derivative lacking a carbamide fragment. |